Dr. Fayez Bahamad Jr

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(61) 3328-6009

Livro e Artigos Científicos

 

 

Total: 22. Listados de 1 a 20.

 

 

Enxaqueca Associada a Disfunsão Auditivo Vestibular
Renato Cal, Fayez Bahmad Jr

Revista Brasileira de Otorrinolaringologia 74 (4) Julho/Agosto 2008


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Revista Gestão e Saúde - Militares e Ruído
Monique Antunes de Souza Chelminski Barreto, Fayez Bahmad Júnior

Revista Eletrônica Gestão & Saúde  Vol.03, Nº. 03, Ano 2012:p. 1094-108


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ABSTRACT

Exposure to noise can produce a deleterious effect on hearing health and may cause Hearing Loss Induced by High Levels of Sound Pressure or an acoustic trauma, by noise continuous exposition or impact noise. Objective: Review studies on hearing loss in military in respect of hearing assessment and the type of noise exposure. Method: Search was conducted in the databases MEDLINE, LILACS, SCIELO, BIREME, PUBMED, COCHRANE, CAPES and textbooks, using the descriptors: hearing loss, occupational noise, military, hearing assessment and their related terms in the English language, in studies published from 2000 to 2010. Results: It was noted that the procedures more used in hearing assessment were evoked otoacoustic emissions and threshold tonal audiometry and high frequency audiometry; as regards of noise type, in the Army, the exposition to noise impact is greater in practice shooting, while in the Navy and Aeronautics, prevails the aircraft and vessels continuous noise. Conclusion: It is necessary a higher level of awareness about the risks of exposure to noise, as well as elaboration and deployment of Hearing Conservation Programs for the military.

 

Histopathology of Ossicular Grafts and Implants in Chronic Otitis Media
Fayez Bahmad, Saumil N Merchant

The Annals of otology, rhinology, and laryngology. 04/2007; 116(3):181-91. · 1.05 Impact Factor


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ABSTRACT

We describe the histopathology of ossicular grafts and implants so as to provide insight into factors that may influence functional results after surgery for chronic otitis media. Histopathologic observations were made on 56 cases: 50 surgical specimens and 6 temporal bone cases in which the graft was sectioned in situ. Autogenous malleus, incus, and cortical bone grafts behaved in a similar manner and maintained their morphological size, shape, and contour for extended periods of time, at least up to 30 years. These histopathologic observations support the continued use of autograft ossicular and cortical bone grafts for middle ear reconstruction. Cartilage grafts developed chondromalacia with resulting loss of stiffness and showed a tendency to undergo resorption. Synthetic prostheses made of porous plastic (Plastipore, Polycel) elicited foreign body giant cell reactions with various degrees of biodegradation of the implants. Prostheses made of hydroxyapatite and Bioglass were enveloped by a lining of connective tissue and mucosal epithelium. The Bioglass material was broken down into small fragments and partially resorbed by a host response within the middle ear. These results warrant caution in the use of prostheses made of porous plastic or Bioglass.

 

Histopathology of Ossicular Grafts and Implants in Chronic Otitis Media
Fayez Bahmad Jr, Saumil N. Merchant


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ABSTRACT

Objectives-We describe the histopathology of ossicular grafts and implants so as to provide insight into factors that may influence functional results after surgery for chronic otitis media.
Methods-Histopathologic observations were made on 56 cases: 50 surgical specimens and 6 temporal bone cases in which the graft was sectioned in situ. Results and Conclusions-Autogenous malleus, incus, and cortical bone grafts behaved in a similar manner and maintained their morphological size, shape, and contour for extended periods of time, at least up to 30 years. These histopathologic observations support the continued use of autograft ossicular and cortical bone grafts for middle ear reconstruction. Cartilage grafts developed chondromalacia with resulting loss of stiffness and showed a tendency to undergo resorption. Synthetic prostheses made of porous plastic (Plastipore, Polycel) elicited foreign body giant cell reactions with various degrees of biodegradation of the implants. Prostheses made of hydroxyapatite and Bioglass were enveloped by a lining of connective tissue and mucosal epithelium. The Bioglass material was broken down into small fragments and partially resorbed by a host response within the middle ear. These results warrant caution in the use of prostheses made of porous plastic or Bioglass.

 

Locus for Familial Migrainous Vertigo Disease Maps to Chromosome 5q35
Fayez Bahmad, Steven R DePalma, Saumil N Merchant, Roberta L Bezerra, Carlos A Oliveira, Christine E

The Annals of otology, rhinology, and laryngology. 09/2009; 118(9):670-6. · 1.05 Impact Factor


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ABSTRACT

Migrainous vertigo (episodic vertigo associated with migraine) is sometimes inherited as an autosomal dominant trait. However, neither disease genes nor loci that might be responsible have been reported. We sought to map the genetic locus for familial migrainous vertigo in a 4-generation family and to define the progression of disease in this family. We studied 23 members in a family in whom migrainous vertigo was inherited as an autosomal dominant trait. Clinical information obtained included case histories and results of otolaryngological, neurologic, audiometric, and imaging evaluations. Genome-wide linkage analysis was performed with Affymetrix Genechip Human Mapping 10K microarrays. Genotyping of family members´ DNA with microsatellite markers was used to further assess candidate loci identified from the whole-genome scan. Of 23 family members, 10 suffered from migrainous vertigo beginning after 35 years of age. Migraine headaches usually preceded the onset of vertigo by 15 to 20 years. Longitudinal audiometric studies over 12 years showed stable, high-frequency sensorineural hearing loss consistent with presbycusis. Low-frequency or fluctuating hearing loss was not observed. The results of vestibular testing and imaging studies were unremarkable. Genetic analysis defined a 12.0 MB interval on chromosome 5q35 between loci rs244895 and D5S2073 that contained the disease gene (logarithm of odds score, 4.21). We report the first locus for familial migrainous vertigo, which mapped to 5q35

 

Intratympanic Dexamethasone Injections as a Treatment for Severe, Disabling Tinnitus
Mercedes F S Araújo, Carlos A Oliveira, Fayez M Bahmad

Archives of otolaryngology--head & neck surgery. 03/2005; 131(2):113-7. · 1.63 Impact Factor


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ABSTRACT

To test the effectiveness of intratympanic dexamethasone injections as a treatment for severe disabling cochlear tinnitus. Randomized, prospective, single-blind study. Academic tertiary referral hospital. Thirty-six patients with severe disabling tinnitus predominantly of cochlear origin were randomly assigned to receive intratympanic injections of a dexamethasone solution or isotonic sodium chloride (saline) solution. Under topical anesthesia and after randomization, 36 patients received 0.5-mL intratympanic injections once per week for 4 weeks of either a 4-mg/mL dexamethasone solution or saline solution. Five patients were excluded from analysis because they did not complete the treatment or did not return for follow-up. Improvement of tinnitus measured with a visual analog scale. The 2 groups were similar in age, sex, tinnitus laterality, measurement of tinnitus intensity on the visual analog scale, and main otologic diagnosis. We considered a 2-point improvement on the visual analog scale to be significant. Twenty-nine percent of the ears in the saline group and 33% of the ears in the dexamethasone group showed significant improvement immediately after completion of treatment. These measurements were not significantly different from each other. Follow-up varied from 13 to 31 months, and the patients with improved tinnitus returned to the initial measurements over time. There was no advantage in intratympanic injections of dexamethasone over saline solution in the treatment of severe, disabling tinnitus. Both solutions produced a placebolike improvement.

 

Psychological Factors in Severe Disabling Tinnitus
Mercedes F. S. Araújo, Carlos A. Oliveira, Fayez M. Bahmad, Jr, James J. McMillan,

ARCH OTOLARYNGOL HEAD NECK SURG/VOL 131, SEP 2005


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Journal Article: Radiology Quiz Case 1
Jairo Barros Filho, Fayez Bahmad, Carlos A C P Oliveira

Archives of otolaryngology--head & neck surgery. 03/2010; 136(3):306. · 1.92 Impact Factor


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Radiology Quiz Case 1: Diagnosis

ARCH OTOLARYNGOL HEAD NECK SURG/VOL 130, FEB 2004


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Potencial evocado miogênico vestibular: uma visão geral
Renato Cal, Fayez Bahmad Jr.

BRAZILIAN JOURNAL OF OTORHINOLARYNGOLOGY 75 (3) MAIO/JUNHO 2009


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Surdez Neurossensorial de Origem Infecciosa
Carlos A. Oliveira, Fayez Bahmad Jr., André Luís Lopes Sampaio, Mercedes Fabiana dos Santos Araújo


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Otopathology in Mohr-Tranebjaerg Syndrome
Fayez Bahmad Jr, Saumil N. Merchant, Joseph B. Nadol Jr, Lisbeth Tranebjærg


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OTOPATHOLOGY IN MOHR-TRANEBJÆRG SYNDROME
Fayez Bahmad Jr MD, Saumil N. Merchant MD, Joseph B. Nadol Jr MD, PhD Lisbth Tranebjærg MD

The Laryngoscope. 06/2007; 117(7):1202 - 1208. · 1.75 Impact Factor


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ABSTRACT

Background: Mohr-Tranebjærg syndrome (MTS) is an X-linked, recessive, syndromic sensorineural hearing loss (HL) characterized by onset of deafness in childhood followed later in adult life by progressive neural degeneration affecting the brain and optic nerves. MTS is caused by mutations in the DDP/TIMM8A gene, which encodes for a 97 amino acid polypeptide; this polypeptide is a translocase of the inner mitochondrial membrane.Objectives: To describe the otologic presentation and temporal bone histopathology in four affected individuals with MTS.Material and Methods: All four subjects belonged to a large, multigenerational Norwegian family and were known to carry a frame shift mutation in the TIMM8A gene. Temporal bones were removed at autopsy and studied by light microscopy. Cytocochleograms were constructed for hair cells, stria vascularis, and cochlear neuronal cells. Vestibular neurons were also counted.Results: All four subjects developed progressive HL in early childhood, becoming profoundly deaf by the age of 10 years. All four developed language, and at least one subject used amplification in early life. Audiometric evaluation in two subjects showed 80- to 100-dB HL by the age of 10 years. The subjects died between the ages of 49 and 67. The otopathology was strikingly similar in that all bones examined showed near-total loss of cochlear neuronal cells and severe loss of vestibular neurons. When compared with age-matched controls, there was 90% to 95% loss of cochlear neurons and 75% to 85% loss of vestibular neurons.Conclusions: We infer that the HL in MTS is likely to be the result of a postnatal and progressive degeneration of cochlear neurons and that MTS constitutes a true auditory neuropathy. Our findings have implications for clinical diagnosis of patients with MTS and management of the HL.

 

Histopathology of Nonsyndromic Autosomal Dominant Midfrequency Sensorineural Hearing Loss
Fayez Bahmad, Jennifer O'Malley, Lisbeth Tranebjaerg, Saumil N Merchant

Otology & neurotology : official publication of the American Otological Society, American Neurotolog


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ABSTRACT

Autosomal dominant, nonsyndromic, midfrequency sensorineural hearing loss (SNHL) is a well-known clinical entity. There are no reported histopathologic studies of temporal bones from individuals with such a hearing loss. To describe the otopathology in 2 affected individuals from 2 different kindreds with nonsyndromic, dominant, midfrequency SNHL. Both subjects belonged to multigenerational families with nonsyndromic, autosomal dominant SNHL showing a cookie-bite pattern. Temporal bones were removed at autopsy and studied by light microscopy. Cytocochleograms were constructed for hair cells, stria vascularis, and cochlear neuronal cells. Subject 1 (a 77-yr-old man) from Kindred A was diagnosed in early childhood with an SNHL that was progressive, reaching profound levels by adulthood. Both cochleae showed complete loss of inner and outer hair cells, moderate to severe diffuse atrophy of the stria vascularis, and severe loss of cochlear neurons, including the peripheral dendrites. The hearing loss in Subject 2 (an 82-yr-old man from Kindred B) began in late childhood, was slowly progressive, and involved the higher frequencies later in life. Histopathology showed loss of outer and inner hair cells in the basal turn of the cochlea, moderate to severe loss of stria vascularis, but relative preservation of peripheral dendrites and cochlear neurons. The main histopathologic abnormalities were loss of hair cells, stria vascularis, and cochlear neurons in 1 case and loss of hair cells and stria vascularis in the second case. Our results are consistent with the hypothesis that dysfunction and loss of hair cells may have been the primary histopathologic correlate for the midfrequency hearing losses in these 2 subjects.

 

Temporal Bone Histolopathology Case of the Month Paget Disease of the Temporal Bone
Fayez Bahmad, Jr. and Saumil N. Merchant

Otology & Neurotology, Vol. 28, No. 8, 2007


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Paget Disease of the Temporal Bone
Fayez Bahmad, Saumil N Merchant

Otology & neurotology : official publication of the American Otological Society, American Neurotolog


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Pendred Syndrome in a Large Consanguineous Brazilian Family Caused by a Homozygous Mutation in the SLC26A4
Adriana Lofrano-Porto, Gustavo B Barra, Paula P Nascimento, Patrícia G G Costa, Erica C Garcia, Rod

Arquivos brasileiros de endocrinologia e metabologia. 12/2008; 52(8):1296-303. · 0.68 Impact Factor


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ABSTRACT

Pendred Syndrome (PS) is an autossomal recessive disorder characterized by sensorineural deafness, goiter and iodide organification defect. The hearing loss is associated with inner ear abnormalities, ranging from an isolated enlarged vestibular aqueduct (EVA) to a typical coclear dysplasia. Mutations in the gene that encodes pendrin (SLC26A4), a chloride/iodide transporter, have been shown to be associated with PS. We describe the clinical and molecular characteristics of a large consanguineous family harboring a mutation in the SLC26A4 gene. The proband was a 26-year-old deaf Brazilian woman who presented a bulky multinodular goiter and hypothyroidism since puberty. Five other siblings were deaf: one brother had a similar phenotype, three siblings also had goiters but normal thyroid function tests, and one brother had only a subtle thyroid enlargement. Other 4 siblings had no thyroid or hearing disorder. Parents were first degree cousins and had normal hearing. The mother was healthy, except for subclinical hypothyroidism; the father was deceased. A perchlorate test in the proband showed a discharge of 21% of the incorporated iodide 2h after the administration of 1g of KClO4. Audiological examinations showed profound hearing loss in all deaf subjects; CT and MRI of the temporal bones showed EVA in all of them. Genomic DNA was isolated from whole blood, from the 6 affected and 4 unaffected siblings, the mother and control. The coding region of the PDS gene (exons 2-21), including exon/intron boundaries, were amplified by PCR and sequenced. A single base-pair (T) deletion at position 1197 of exon 10 was detected in homozygous state in the 6 deaf siblings. The mother and 2 unaffected siblings were heterozygous for this mutation, which has been described by Everett et al. The 1197delT mutation is predicted to result in a frameshift and a truncated protein. The existence of PS phenocopies and intrafamilial phenotypic variability are well documented. The definite diagnosis requires molecular analysis. Our study illustrates the value and challenges of mutational analysis in selected patients with PS.

 

Intratympanic Methylprednisolone as Rescue Therapy in Sudden Sensorineural Hearing Loss
Igor Teixeira Raymundo, Fayez Bahmad Jr, Jairo Barros Filho, Thaís Gonçalves Pinheiro, Nilda Agostin


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ABSTRACT

Treatment in sudden sensorineural hearing loss is a contentious issue, today, oral steroids are the most common choice and considered the best treatment option, but the use of intratympanic steroids has become an attractive alternative, especially in cases when systemic therapy fails, or to avoid the side effects of the systemic use of steroids. Aim: To describe the results of  intratympanic methylprednisolone in idiopathic sudden sensorineural hearing loss after failure of oral prednisolone.
Methods: In a prospective study fourteen patients with idiopathic sudden sensorineural hearing loss were treated with intratympanic methylprednisolone after failing in the treatment with systemic steroids. Pretreatment and post-treatment audiometric evaluations including pure tone average (PTA) and speech reception thresholds (SRT) were analyzed. Results: Ten from 14 patients treated with intra-tympanic methylprednisolone presented with hearing recovery > 20 dB in PTA or 20% in SRT.
Conclusion: Three intratympanic injections of methylprednisolone improved pure-tone average or speech discrimination scores for a subset of sudden hearing loss subjects that failed to benefit from oral steroids.

 

Histopatologia do osso temporal: surdez súbita idiopática
Fayez Bahmad Jr

REVISTA BRASILEIRA DE OTORRINOLARINGOLOGIA 74 (1) JANEIRO/FEVEREIRO 2008


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Revisão de Estapedotomia com uso de 2-Cianobutilacrilato Fixando Prótese de Teflon-platina a Bigorna
Carlos Lúcio Pinto Vieira Filho, Fayez Bahmad Júnior, Heitor Dantas Guerreiro, Kléber Alberto de Sou

Arq. Int. Otorrinolaringol. / Intl. Arch. Otorhinolaryngol., São Paulo, v.11, n.2, p. 202-206, 2007.


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